FlamLeve FAQ

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  • What is FlamLeve™?

    FlamLeve™ is a natural anti-inflammatory agent that helps to reduce pain by combatting inflammation. FlamLeve™ contains a unique blend of herbal extracts that complement each other and work together to moderate inflammatory pathways in the body. The combined effects help to alleviate the pain and discomfort caused by inflammation and offers protection against inflammatory damage to tissues, joints and organs.

  • What is the dosage for FlamLeve™?

    Take 1-3 tablets per day as needed.

  • What are the side effects of FlamLeve™?

    Extensive clinical trials in human subjects have demonstrated that the natural ingredients contained in FlamLeve™ have a gentle anti-inflammatory effect with a low side-effect profile. These pose a low health risk, especially when compared to pharmaceutical anti-inflammatory and painkilling drugs. Mild gastrointestinal irritation may occur.  To avoid this potential effect, FlamLeve™ should always be taken with food.

  • Where can I buy FlamLeve™?

    • Shop Online.
    • South Africa: At any leading pharmacy. No prescription is required.
    • Namibia: At any leading pharmacy. No prescription is required.
    • International: Contact us at info@mnilifestyle.co.za for international purchasing information.

  • Will my medical aid pay for FlamLeve™?

    FlamLeve™ has a NAPPI code, which means that most medical aids will offer reimbursement, providing you have a savings option and available funds.

  • What is the difference between FlamLeve™ and RheumaLin™?

    FlamLeve™ is your everyday anti-inflammatory, ideally used to relieve mild pain, discomfort and stiffness resulting from muscle injury and inflammation, as well as an assistive therapy for acute and chronic inflammatory conditions.

    RheumaLin™, on the other hand, is specifically formulated to address common causes of chronic inflammatory pain relating specifically to the spine (back & neck), hands, hip and knee joints.

    Since the ingredients in FlamLeve™ and RheumaLin™ complement each other, they can be used in conjunction at the same time for quicker results.

  • What is inflammation and why is it bad?

    Inflammation is a natural process controlled by your immune system, and responsible for removing and repairing damaged tissue, as well as attacking invading organisms like bacteria and viruses. Under normal circumstances, inflammation is therefore a good thing, but if it becomes overactive, can lead to pain and disease.

    Acute inflammation, like swelling that happens in response to an injury, is not a bad thing in itself. It can, however, cause a lot of discomfort, stiffness and pain. It can also sometimes slow recovery if it gets out of hand. Taking anti-inflammatories can therefore be useful to both your comfort and recovery.

    When inflammation becomes chronic, it starts to cause a long list of issues, and eventually contributes to the development of many chronic diseases. This occurs through multiple mechanisms. Firstly, your immune system may lose the ability to tell the difference between healthy and diseased tissue. What this leads to is inflammation which starts to attack healthy cells and organs, resulting in a wide range of disorders, from diabetes, heart disease, arthritis and cancer to autoimmune conditions like lupus. There is new evidence that systemic, chronic inflammation even leads to neurological disorders, including Alzheimer’s disease. In order to protect your long term health it is therefore important to be aware of, and actively manage inflammation.

  • What is the scientific explanation of why inflammation can cause damage?

    Inflammation – the biological equivalent of rust

    Proteolysis is the scientific term for the breakdown of proteins into smaller molecules such a peptides and  amino acids. A key feature of the inflammatory response is the production of several protein-dissolving or proteolytic enzymes.  Collectively, these display a remarkable capacity to dissolve virtually any protein-based structure through powerful enzymatic activity, often at an astonishing rate. Proteolytic activity cannot be selectively focussed to a specific region, however, and as a result, collateral damage to structurally sound tissues commonly occurs.

    When proteolytic activity dissolves the protein-based components of injured tissue in preparation for removal, the benefits are obvious. However, when healthy, protein-containing tissue such as cartilage, tendons, intervertebral disks, ligaments and bones are inadvertently degraded through the same process, a disease state ensues. Unfortunately, the rate of damage caused by the inflammatory process frequently outweighs the rate of repair resulting in a net loss.

    Progressive damage caused by inflammation

    In the beginning, the degenerative activities of inflammation are neither visible nor quantifiable. However, over time the evidence for degeneration becomes both visibly obvious and measurable, when various distorted and structurally impaired anatomical components of the spine or joints are easily identifiable on X-ray or scan.

    What activates, regulates and ultimately deactivates the inflammatory process is a vast topic that has attracted significant scientific interest over the last decade. Supported by the recent explosion of molecular information gained from various novel technology platforms, scientists have only recently begun to unravel the immense complexity of molecular behaviour at this level.

    The molecular benefits of FlamLeve™ against inflammation

    In depth research has been conducted on the molecular effects of several of the medicinal compounds present in both curcumin and Boswellia bark extract. Studies have indicated that these demonstrate multimodal molecular activity via several biological targets involved in the activation and acceleration of inflammation via the activation of proteolytic inflammatory enzymes and molecular pain pathways.

    Proteolytic inflammatory enzymes that FlamLeve™ include the Matrix metallopeptidases (MMPs), cathepsins (CatG) and prostaglandin E2 synthase-1 (mPGES-1), all powerful promotors  of progressive tissue destruction. Molecular pain pathways involve the reduction in the levels the pain producing cytokine TNF-alpha, achieved through the inhibition of NF-κβ based pro-inflammatory activation.

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